Michael Lewis
Michael P. Lewis is a former Fulbright Scholar and an aspiring physician-scientist. As a Research Assistant II in Voit Laboratory, he is primarily focused on developing CRISPR-Cas9-based stem cell lines, in vitro, and introducing them into genetically-engineered mouse models in vivo, to study how various genetic perturbations affect normal human hematopoiesis and cancerous leukemogenesis.
He completed his B.S. degrees in chemistry and biology at the University of Texas at Arlington (UTA) and the University of North Texas (UNT) and has prior experience in higher education, the pharmaceutical industry, basic scientific research, and the healthcare field.
Before joining Voit Lab, Michael had the opportunity to research under Dr. Cheng-Ming Chiang in the Simmons Comprehensive Cancer Center (SCCC) at UT Southwestern, studying the key epigenetic regulator and transcription factor bromodomain-containing protein 4 (BRD4) and its isoform-specific roles in PyMT-driven mouse breast cancer and normal mammalian physiology.
Outside of the lab, Michael enjoys playing piano, trying new cuisines, and learning foreign languages.
